Our model shows how disinformation spreads on a random network of individuals. The network is weighted and directed. We are looking at how different factors affect how fast, or how many people get “infected” with the misinformation. One of the main factors that we were curious about was perceived trustworthiness. This is because we want to see if people of power, or a high degree of perceived trustworthiness, were able to push misinformation to more people and convert more people to believe the information.

A curious aspect of the Covid-19 pandemic is the clustering of outbreaks. Evidence suggests that 80\% of people who contract the virus are infected by only 19% of infected individuals, and that the majority of infected individuals faile to infect another person. Thus, the dispersion of a contagion, $k$, may be of more use in understanding the spread of Covid-19 than the reproduction number, R0.

The Virus Transmission with Super-spreaders model, written in NetLogo, is an adaptation of the canonical Virus Transmission on a Network model and allows the exploration of various mitigation protocols such as testing and quarantines with both homogenous transmission and heterogenous transmission.

The model consists of a population of individuals arranged in a network, where both population and network degree are tunable. At the start of the simulation, a subset of the population is initially infected. As the model runs, infected individuals will infect neighboring susceptible individuals according to either homogenous or heterogenous transmission, where heterogenous transmission models super-spreaders. In this case, k is described as the percentage of super-spreaders in the population and the differing transmission rates for super-spreaders and non super-spreaders. Infected individuals either recover, at which point they become resistant to infection, or die. Testing regimes cause discovered infected individuals to quarantine for a period of time.

Multi-layer network agent-based model of the progression of the COVID19 infection, digital contact tracing

This model implements two types of network diffusion from an initial group of activated nodes. In complex contagion, a node is activated if the proportion of neighbour nodes that are already activated exceeds a given threshold. This is intended to represented the spread of health behaviours. In simple contagion, an activated node has a given probability of activating its inactive neighbours and re-tests each time step until all of the neighbours are activated. This is intended to represent information spread.

A range of networks are included with the model from secondary school friendship networks. The proportion of nodes initially activated and the method of selecting those nodes are controlled by the user.

This is a basic Susceptible, Infected, Recovered (SIR) model. This model explores the spread of disease in a space. In particular, it explores how changing assumptions about the number of susceptible people, starting number of infected people, as well as the disease’s infection probability, and average duration of infection. The model shows that the interactions of agents can drastically affect the results of the model.

We used it in our course on COVID-19: https://www.csats.psu.edu/science-of-covid19

This is an extension of the basic Suceptible, Infected, Recovered (SIR) model. This model explores the spread of disease in two spaces, one a treatment, and one a control. Through the modeling options, one can explore how changing assumptions about the number of susceptible people, starting number of infected people, the disease’s infection probability, and average duration impacts the outcome. In addition, this version allows users to explore how public health interventions like social distancing, masking, and isolation can affect the number of people infected. The model shows that the interactions of agents, and the interventions can drastically affect the results of the model.

We used the model in our course about COVID-19: https://www.csats.psu.edu/science-of-covid19

We study cultural dissemination in the context of an Axelrod-like agent-based model describing the spread of cultural traits across a society, with an added element of social influence. This modification produces absorbing states exhibiting greater variation in number and size of distinct cultural regions compared to the original Axelrod model, and we identify the mechanism responsible for this amplification in heterogeneity. We develop several new metrics to quantitatively characterize the heterogeneity and geometric qualities of these absorbing states. Additionally, we examine the dynamical approach to absorbing states in both our Social Influence Model as well as the Axelrod Model, which not only yields interesting insights into the differences in behavior of the two models over time, but also provides a more comprehensive view into the behavior of Axelrod’s original model. The quantitative metrics introduced in this paper have broad potential applicability across a large variety of agent-based cultural dissemination models.

EMMIT is an end-user developed agent-based simulation of malaria transmission. The simulation’s development is a case study demonstrating an approach for non-technical investigators to easily develop useful simulations of complex public health problems. We focused on malaria transmission, a major global public health problem, and insecticide resistance (IR), a major problem affecting malaria control. Insecticides are used to reduce transmission of malaria caused by the Plasmodium parasite that is spread by the Anopheles mosquito. However, the emergence and spread of IR in a mosquito population can diminish the insecticide’s effectiveness. IR results from mutations that produce behavioral changes or biochemical changes (such as detoxification enhancement, target site alterations) in the mosquito population that provide resistance to the insecticide. Evolutionary selection for the IR traits reduces the effectiveness of an insecticide favoring the resistant mosquito population. It has been suggested that biopesticides, and specifically those that are Late Life Acting (LLA), could address this problem. LLA insecticides exploit Plasmodium’s approximate 10-day extrinsic incubation period in the mosquito vector, a delay that limits malaria transmission to older infected mosquitoes. Since the proposed LLA insecticide delays mosquito death until after the exposed mosquito has a chance to produce several broods of offspring, reducing the selective pressure for resistance, it delays IR development and gives the insecticide longer effectivity. Such insecticides are designed to slow the evolution of IR thus maintaining their effectiveness for malaria control. For the IR problem, EMMIT shows that an LLA insecticide could work as intended, but its operational characteristics are critical, primarily the mean-time-to-death after exposure and the associated standard deviation. We also demonstrate the simulation’s extensibility to other malaria control measures, including larval source control and policies to mitigate the spread of IR. The simulation was developed using NetLogo as a case study of a simple but useful approach to public health research.

The Netlogo model is a conceptualization of the Moria refugee camp, capturing the household demographics of refugees in the camp, a theoretical friendship network based on values, and an abstraction of their daily activities. The model then simulates how Covid-19 could spread through the camp if one refugee is exposed to the virus, utilizing transmission probabilities and the stages of disease progression of Covid-19 from susceptible to exposed to asymptomatic / symptomatic to mild / severe to recovered from literature. The model also incorporates various interventions - PPE, lockdown, isolation of symptomatic refugees - to analyze how they could mitigate the spread of the virus through the camp.

The Episim framework builds upon the established transportation simulation MATSim and is capable of tracking agents’ movements within a network and thus computing infection chains. Several characteristics of the virus and the environment can be parametred, whilst the infection dynamics is computed based upon a compartment model. The spread of the virus can be mitigated by restricting the agents’ activity in certain places.